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Institut für Pharmakologie am Universitätsklinikum Essen
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Hufelandstr. 55
45122 Essen
45122 Essen
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Lehrbeauftragte/r, Institut für Pharmakologie
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Die folgenden Publikationen sind in der Online-Universitätsbibliographie der Universität Duisburg-Essen verzeichnet. Weitere Informationen finden Sie gegebenenfalls auch auf den persönlichen Webseiten der Person.
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Phosphodiesterase 8 governs cAMP/PKA-dependent reduction of L-type calcium current in human atrial fibrillation : a novel arrhythmogenic mechanismIn: European Heart Journal Jg. 44 (2023) Nr. 27, S. 2483 - 2494Online Volltext: dx.doi.org/ (Open Access)
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Kv1.1 potassium channel subunit deficiency alters ventricular arrhythmia susceptibility, contractility, and repolarizationIn: Physiological Reports Jg. 9 (2021) Nr. 1, e14702Online Volltext: dx.doi.org/ (Open Access)
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CaMKII activity contributes to homeometric autoregulation of the heart : A novel mechanism for the Anrep effectIn: Journal of Physiology (2020)Online Volltext: dx.doi.org/ Online Volltext (Open Access)
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German Cardiac Society Working Group on Cellular Electrophysiology state-of-the-art paper : impact of molecular mechanisms on clinical arrhythmia managementIn: Clinical Research in Cardiology Jg. 108 (2019) Nr. 6, S. 577 - 599Online Volltext: dx.doi.org/ (Open Access)
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Muscarinic type-1 receptors contribute to IK,ACh in human atrial cardiomyocytes and are upregulated in patients with chronic atrial fibrillationIn: International Journal of Cardiology Jg. 255 (2018) S. 61 - 68Online Volltext: dx.doi.org/ Online Volltext (Open Access)
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Inverse remodelling of K2P 3.1 K+ channel expression and action potential duration in left ventricular dysfunction and atrial fibrillation : Implications for patient-specific antiarrhythmic drug therapyIn: European Heart Journal Jg. 38 (2017) Nr. 22, S. 1764 - 1774Online Volltext: dx.doi.org/
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Nucleoside Diphosphate Kinase-C Suppresses cAMP Formation in Human Heart FailureIn: Circulation Jg. 135 (2017) Nr. 9, S. 881 - 897Online Volltext: dx.doi.org/ (Open Access)
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The inward rectifier current inhibitor PA-6 terminates atrial fibrillation and does not cause ventricular arrhythmias in goat and dog modelsIn: British Journal of Pharmacology (BJP) Jg. 174 (2017) Nr. 15, S. 2576 - 2590Online Volltext: dx.doi.org/ (Open Access)
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Atrial-Selective Potassium Channel BlockersIn: Cardiac Electrophysiology Clinics Jg. 8 (2016) Nr. 2, S. 411 - 421Online Volltext: dx.doi.org/
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Computational models of atrial cellular electrophysiology and calcium handling, and their role in atrial fibrillationIn: Journal of Physiology Jg. 594 (2016) Nr. 3, S. 537 - 553Online Volltext: dx.doi.org/
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Dysfunction of the β2-spectrin-based pathway in human heart failureIn: American Journal of Physiology: Heart and Circulatory Physiology Jg. 310 (2016) Nr. 11, S. H1583 - H1591Online Volltext: dx.doi.org/ (Open Access)
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Response to Letter Regarding Article, "Upregulation of K2P3.1 K+ Current Causes Action Potential Shortening in Patients With Chronic Atrial Fibrillation"In: Circulation Jg. 133 (2016) Nr. 11, S. e440 - 441Online Volltext: dx.doi.org/ Online Volltext (Open Access)
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S-glutathiolation impairs phosphoregulation and function of cardiac myosin-binding protein C in human heart failureIn: The FASEB Journal: the official journal of the Federation of American Societies for Experimental Biology / Experimental Biology ; Transforming the future through science ; Chicago, April 22-26, 2017 Jg. 30 (2016) Nr. 5, S. 1849 - 1864Online Volltext: dx.doi.org/ Online Volltext (Open Access)
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The combined effects of ranolazine and dronedarone on human atrial and ventricular electrophysiologyIn: Journal of Molecular and Cellular Cardiology Jg. 94 (2016) S. 95 - 106Online Volltext: dx.doi.org/
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Alterations in the interactome of serine/threonine protein phosphatase type-1 in atrial fibrillation patientsIn: Journal of the American College of Cardiology Jg. 65 (2015) Nr. 2, S. 163 - 173Online Volltext: dx.doi.org/ (Open Access)
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Application of the RIMARC algorithm to a large data set of action potentials and clinical parameters for risk prediction of atrial fibrillationIn: Medical and Biological Engineering and Computing Jg. 53 (2015) Nr. 3, S. 263 - 273Online Volltext: dx.doi.org/ (Open Access)
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Atrial Fibrillation Activates AMP-Dependent Protein Kinase and its Regulation of Cellular Calcium Handling : Potential Role in Metabolic Adaptation and Prevention of ProgressionIn: Journal of the American College of Cardiology Jg. 66 (2015) Nr. 1, S. 47 - 58Online Volltext: dx.doi.org/ (Open Access)
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Calcium Handling Abnormalities as a Target for Atrial Fibrillation Therapeutics : How Close to Clinical Implementation?In: Journal of Cardiovascular Pharmacology Jg. 66 (2015) Nr. 6, S. 515 - 522Online Volltext: dx.doi.org/
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Dysfunction in the βII spectrin-dependent cytoskeleton underlies human arrhythmiaIn: Circulation Jg. 131 (2015) Nr. 8, S. 695 - 708Online Volltext: dx.doi.org/ (Open Access)
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Expression and function of Kv1.1 potassium channels in human atria from patients with atrial fibrillationIn: Basic Research in Cardiology Jg. 110 (2015) Nr. 5, S. 47Online Volltext: dx.doi.org/ Online Volltext (Open Access)
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Identification of microRNA-mRNA dysregulations in paroxysmal atrial fibrillationIn: International Journal of Cardiology Jg. 184 (2015) S. 190 - 197Online Volltext: dx.doi.org/ Online Volltext (Open Access)
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Methods for isolating atrial cells from large mammals and humansIn: Journal of Molecular and Cellular Cardiology Jg. 86 (2015) S. 187 - 198Online Volltext: dx.doi.org/ (Open Access)
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New antiarrhythmic targets in atrial fibrillationIn: Future Cardiology Jg. 11 (2015) Nr. 6, S. 645 - 654Online Volltext: dx.doi.org/
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Upregulation of K(2P)3.1 K+ Current Causes Action Potential Shortening in Patients With Chronic Atrial FibrillationIn: Circulation Jg. 132 (2015) Nr. 2, S. 82 - 92Online Volltext: dx.doi.org/ (Open Access)
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Calcium dysregulation in atrial fibrillation : the role of CaMKIIIn: Frontiers in Pharmacology Jg. 5 (2014) S. 30Online Volltext: dx.doi.org/ (Open Access)
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Cardiac safety assaysIn: Current Opinion in Pharmacology Jg. 15 (2014) Nr. 1, S. 16 - 21Online Volltext: dx.doi.org/
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Cellular and molecular electrophysiology of atrial fibrillation initiation, maintenance, and progressionIn: Circulation Research Jg. 114 (2014) Nr. 9, S. 1483 - 1499Online Volltext: dx.doi.org/
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Cellular and molecular mechanisms of atrial arrhythmogenesis in patients with paroxysmal atrial fibrillationIn: Circulation Jg. 129 (2014) Nr. 2, S. 145 - 156Online Volltext: dx.doi.org/
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GW25-e5168 Impaired Post-Transcriptional Regulation of RyR2 by microRNA-106b-25 Cluster Promotes Atrial FibrillationIn: Journal of the American College of Cardiology Jg. 64 (2014) Nr. 16 Suppl., S. C59Online Volltext: dx.doi.org/
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Impaired local regulation of ryanodine receptor type 2 by protein phosphatase 1 promotes atrial fibrillationIn: Cardiovascular Research Jg. 103 (2014) Nr. 1, S. 178 - 187Online Volltext: dx.doi.org/ Online Volltext (Open Access)
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Kardiovaskuläre Pharmakotherapie : Risiken und NebenwirkungenIn: Herz: Cardiovascular Diseases Jg. 39 (2014) Nr. 2, S. 227 - 240Online Volltext: dx.doi.org/
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Loss of MicroRNA-106b-25 Cluster Promotes Atrial Fibrillation by Enhancing Ryanodine Receptor Type-2 Expression and Calcium ReleaseIn: Circulation: Arrhythmia and Electrophysiology Jg. 7 (2014) Nr. 6, S. 1214 - 1222Online Volltext: dx.doi.org/
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Ryanodine receptor-mediated calcium leak drives progressive development of an atrial fibrillation substrate in a transgenic mouse modelIn: Circulation Jg. 129 (2014) Nr. 12, S. 1276 - 1285Online Volltext: dx.doi.org/
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Tachycardia-induced silencing of subcellular Ca2+ signaling in atrial myocytesIn: The Journal of Clinical Investigation (JCI) Jg. 124 (2014) Nr. 11, S. 4759 - 4772Online Volltext: dx.doi.org/
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Impaired Na+-dependent regulation of acetylcholine-activated inward-rectifier K+ current modulates action potential rate dependence in patients with chronic atrial fibrillationIn: Journal of Molecular and Cellular Cardiology Jg. 61 (2013) S. 142 - 152Online Volltext: dx.doi.org/
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Isolation of human atrial myocytes for simultaneous measurements of Ca2+ transients and membrane currents.In: Journal of Visualized Experiments (2013) Nr. 77, S. e50235Online Volltext: dx.doi.org/
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MicroRNA29 : a mechanistic contributor and potential biomarker in atrial fibrillationIn: Circulation Jg. 127 (2013) Nr. 14, S. 1466 - 1475Online Volltext: dx.doi.org/
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Mutation E169K in junctophilin-2 causes atrial fibrillation due to impaired RyR2 stabilizationIn: Journal of the American College of Cardiology Jg. 62 (2013) Nr. 21, S. 2010 - 2019Online Volltext: dx.doi.org/ (Open Access)
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NSC23766, a widely used inhibitor of Rac1 activation, additionally Acts as a competitive antagonist at muscarinic acetylcholine receptorsIn: Journal of Pharmacology and Experimental Therapeutics Jg. 347 (2013) Nr. 1, S. 69 - 79Online Volltext: dx.doi.org/
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New directions in antiarrhythmic drug therapy for atrial fibrillationIn: Future Cardiology Jg. 9 (2013) Nr. 1, S. 71 - 88Online Volltext: dx.doi.org/
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Oxidized Ca2+/Calmodulin-Dependent Protein Kinase II Triggers Atrial FibrillationIn: Circulation Jg. 128 (2013) Nr. 16, S. 1748 - 1757Online Volltext: dx.doi.org/
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Rhythm control of atrial fibrillation in heart failureIn: Heart Failure Clinics Jg. 9 (2013) Nr. 4, S. 407 - 415Online Volltext: dx.doi.org/
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The biology of human pulmonary veins : does it help us to better understand AF pathophysiology in patients?In: Heart Rhythm Jg. 10 (2013) Nr. 3, S. 392 - 393Online Volltext: dx.doi.org/
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Prof Niels Voigt talks to Prof Stanley Nattel about advances in atrial fibrillation research and career insightsIn: Cardiovascular Research Jg. 114 (2018) Nr. 8, S. e65Online Volltext: dx.doi.org/ Online Volltext (Open Access)
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Abnormal cellular electrophysiology and calcium handling in iPSC-CMs from a patient with tetralogy of Fallot
84th Annual Meeting of the German Society for Experimental and Clinical Pharmacology and Toxicology (DGPT) and the 20th Annual Meeting of the Association of the Clinical Pharmacology Germany (VKliPha) ; 26 February – 1 March 2018. Göttingen,In: Naunyn-Schmiedeberg's Archives of Pharmacology Jg. 391 (2018) Nr. 1, Supplement, S. S18Online Volltext: dx.doi.org/ -
P678: S-glutathiolation of cMyBP-C reduces its phosphorylation and contributes to impairment of cardiac myocyte contractile functionIn: Cardiovascular Research Jg. 103 (2014) S. S123 - S124Online Volltext: dx.doi.org/ (Open Access)
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The Molecular Pathophysiology of Atrial FibrillationIn: Cardiac Electrophysiology: From Cell to Bedside / Zipes, Douglas P.; Jalife, José; Stevenson, William G. (Hrsg.) 2018, S. 396 - 408Online Volltext: dx.doi.org/
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Cholinergic and Constitutive Regulation of Atrial Potassium ChannelIn: Cardiac Electrophysiology: From Cell to Bedside / Zipes, Douglas P.; Jalife, José 2014, S. 383 - 391Online Volltext: dx.doi.org/
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Constitutive Activity of the Acetylcholine-Activated Potassium Current IK,ACh in CardiomyocytesIn: Pharmacology & Therapeutics of Constitutively Active Receptors / Tao, Ya-Xiong (Hrsg.) 2014, S. 393 - 409Online Volltext: dx.doi.org/
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The Molecular Pathophysiology of Atrial FibrillationIn: Cardiac Electrophysiology: From Cell to Bedside / Zipes, Douglas P.; Jalife, José (Hrsg.) 2013, S. 449 - 458Online Volltext: dx.doi.org/
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Molekulare Pathophysiologie bei Patienten mit Vorhofflimmern : HabilitationsschriftDuisburg, Essen (2013) 1 CD-ROM