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Die folgenden Publikationen sind in der Online-Universitätsbibliographie der Universität Duisburg-Essen verzeichnet. Weitere Informationen finden Sie gegebenenfalls auch auf den persönlichen Webseiten der Person.
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PDCD10 Is a Key Player in TMZ-Resistance and Tumor Cell Regrowth : Insights into Its Underlying Mechanism in Glioblastoma CellsIn: Cells , Jg. 13 2024, Nr. 17, 1442DOI (Open Access)
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Effects of medication intake on the risk of hemorrhage in patients with sporadic cerebral cavernous malformationsIn: Frontiers in Neurology , Jg. 13 2023, 1010170DOI (Open Access)
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Novel Function of Cancer Stem Cell Marker ALDH1A3 in Glioblastoma : Pro-Angiogenesis through Paracrine PAI-1 and IL-8In: Cancers , Jg. 15 2023, Nr. 17, 4422DOI (Open Access)
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Cavernous Malformations : Updates in Surgical Management and BiologyIn: Neurosurgery Clinics of North America , Jg. 33 2022, Nr. 4, S. 449 – 460
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Cerebral cavernous malformations do not fall in the spectrum of PIK3CA-related overgrowthIn: Journal of Neurology, Neurosurgery and Psychiatry , Jg. 93 2022, Nr. 8, S. 808 – 815
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Characterization of Temozolomide Resistance Using a Novel Acquired Resistance Model in Glioblastoma Cell LinesIn: Cancers , Jg. 14 2022, Nr. 9, 2211DOI, Online Volltext (Open Access)
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Ischemia-induced inflammation in arteriovenous malformationsIn: Neurosurgical Focus , Jg. 53 2022, Nr. 1, E3DOI (Open Access)
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Medication intake and hemorrhage risk in patients with familial cerebral cavernous malformationsIn: Journal of Neurosurgery (JNS) , Jg. 137 2022, Nr. 4, S. 1088 – 1094DOI (Open Access)
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Multiple cerebral cavernous malformations : Clinical course of confirmed, assumed and non‐familial diseaseIn: European Journal of Neurology , Jg. 29 2022, Nr. 5, S. 1427 – 1434DOI (Open Access)
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PDCD10 promotes the aggressive behaviors of pituitary adenomas by up-regulating CXCR2 and activating downstream AKT/ERK signalingIn: Aging , Jg. 14 2022, Nr. 15, S. 6066 – 6080DOI (Open Access)
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Cerebral cavernous malformations : Prevalence of cardiovascular comorbidities and allergic diseases compared to the normal populationIn: European Journal of Neurology , Jg. 28 2021, Nr. 6, S. 2000 – 2005DOI (Open Access)
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Health-related quality of life in patients with untreated cavernous malformations of the central nervous systemIn: European Journal of Neurology , Jg. 28 2021, Nr. 2, S. 491 – 499DOI (Open Access)
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Modifiable Cardiovascular Risk Factors in Patients With Sporadic Cerebral Cavernous Malformations : Obesity MattersIn: Stroke , Jg. 52 2021, Nr. 4, S. 1259 – 1264DOI (Open Access)
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DNA promoter methylation of CCM genes in human cerebral cavernous malformations : Importance of confirming MSP data through sequencingIn: European Journal of Medical Genetics (EJMG) , Jg. 63 2020, Nr. 12, 104090
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Hemorrhage from cerebral cavernous malformations: The role of associated developmental venous anomaliesIn: Neurology , Jg. 95 2020, Nr. 1, S. e89 – e96
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PDCD10-Deficiency Promotes Malignant Behaviors and Tumor Growth via Triggering EphB4 Kinase Activity in GlioblastomaIn: Frontiers in Oncology , Jg. 10 2020, S. 1377DOI, Online Volltext (Open Access)
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Pathophysiology of Intracranial Aneurysms : COX-2 Expression, Iron Deposition in Aneurysm Wall, and Correlation With Magnetic Resonance ImagingIn: Stroke , Jg. 51 2020, Nr. 8, S. 2505 – 2513DOI (Open Access)
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TET2 promotor methylation and TET2 protein expression in pediatric posterior fossa ependymomaIn: Neuropathology , Jg. 40 2020, Nr. 2, S. 138 – 143DOI (Open Access)
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The predominant expression of cancer stem cell marker ALDH1A3 in tumor infiltrative area is associated with shorter overall survival of human glioblastomaIn: BMC Cancer , Jg. 20 2020, Nr. 1, 672DOI (Open Access)
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Loss of programmed cell death 10 activates tumor cells and leads to temozolomide-resistance in glioblastomaIn: Journal of Neuro-Oncology , Jg. 141 2019, Nr. 1, S. 31 – 41
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Correlation of the venous angioarchitecture of multiple cerebral cavernous malformations with familial or sporadic disease : A susceptibility-weighted imaging study with 7-Tesla MRIIn: Journal of Neurosurgery (JNS) , Jg. 126 2017, Nr. 2, S. 570 – 577
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EphB4 forward signalling mediates angiogenesis caused by CCM3/PDCD10-ablationIn: Journal of Cellular and Molecular Medicine , Jg. 21 2017, Nr. 9, S. 1848 – 1858DOI (Open Access)
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Outcome after conservative management or surgical treatment for new-onset epilepsy in cerebral cavernous malformationIn: Journal of Neurosurgery (JNS) , Jg. 126 2017, Nr. 4, S. 1303 – 1311
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Study of angiogenic signaling pathways in hemangioblastomaIn: Neuropathology , Jg. 37 2017, Nr. 1, S. 3 – 11
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Activation of multiple angiogenic signaling pathways in hemangiopericytomaIn: Brain Tumor Pathology , Jg. 33 2016, Nr. 3, S. 200 – 208
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Loss of endothelial programmed cell death 10 activates glioblastoma cells and promotes tumor growthIn: Neuro-Oncology , Jg. 18 2016, Nr. 4, S. 538 – 548DOI (Open Access)
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Multiple cerebral arterio-venous malformations : impact of multiplicity and hemodynamics on treatment strategiesIn: Acta Neurochirurgica , Jg. 158 2016, Nr. 12, S. 2399 – 2407
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Solitary Sporadic Cerebral Cavernous Malformations : Risk Factors of First or Recurrent Symptomatic Hemorrhage and Associated Functional ImpairmentIn: World Neurosurgery , Jg. 91 2016, S. 73 – 80
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Associated aneurysms in supratentorial arteriovenous malformations : impact of aneurysm size on haemorrhageIn: Cerebrovascular Diseases , Jg. 39 2015, Nr. 2, S. 122 – 129
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Comments: PTEN/PI3K/Akt/VEGF signaling and the cross talk to KRIT1, CCM2, and PDCD10 proteins in cerebral cavernous malformationsIn: Neurosurgical Review , Jg. 38 2015, Nr. 2, S. 236 – 237
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Downregulation of programmed cell death 10 is associated with tumor cell proliferation, hyperangiogenesis and peritumoral edema in human glioblastomaIn: BMC Cancer , Jg. 15 2015, S. 759 – 770DOI (Open Access)
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Regulation of β1 integrin-Klf2-mediated angiogenesis by CCM proteinsIn: Developmental Cell , Jg. 32 2015, Nr. 2, S. 181 – 190DOI (Open Access)
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Posterior fossa arterio-venous malformations : current multimodal treatment strategies and resultsIn: Neurosurgical Review , Jg. 37 2014, Nr. 4, S. 619 – 628
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Correlation of Aquaporin-1 Water Channel Protein Expression with Tumor Angiogenesis in Human AstrocytomaIn: Anticancer Research , Jg. 33 2013, Nr. 2, S. 609 – 613
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Implications of Dll4-Notch signaling activation in primary glioblastoma multiformeIn: Neuro-Oncology , Jg. 15 2013, Nr. 10, S. 1366 – 1378
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Loss of CCM3 impairs DLL4-Notch signalling: implication in endothelial angiogenesis and in inherited cerebral cavernous malformationsIn: Journal of Cellular and Molecular Medicine , Jg. 17 2013, Nr. 3, S. 407 – 418
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The functional Aquaporin 1 −783G/C-polymorphism is associated with survival in patients with glioblastoma multiformeIn: Journal of Surgical Oncology , Jg. 108 2013, Nr. 7, S. 492 – 498
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The venous angioarchitecture of sporadic cerebral cavernous malformations : a susceptibility weighted imaging study at 7 T MRIIn: Journal of Neurology, Neurosurgery and Psychiatry , Jg. 84 2013, Nr. 2, S. 194 – 201
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Two-hit mechanism in cerebral cavernous malformation? A case of monozygotic twins with a CCM1/KRIT1 germline mutationIn: Neurosurgical Review , Jg. 36 2013, Nr. 3, S. 483 – 486
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Association of the CC genotype of the regulatory BCL2 promoter polymorphism (−938C>A) with better 2-year survival in patients with glioblastoma multiformeIn: Journal of Neurosurgery (JNS) , Jg. 114 2011, Nr. 6, S. 1631 – 1639
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In vitro characterization of the angiogenic phenotype and genotype of the endothelia derived from sporadic cerebral cavernous malformationsIn: Neurosurgery , Jg. 69 2011, Nr. 3, S. 722 – 732
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Role of the GNAS1 T393C polymorphism in patients with glioblastoma multiformeIn: Journal of Clinical Neuroscience , Jg. 18 2011, Nr. 11, S. 1495 – 1499
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Cerebral cavernous malformation protein CCM1 inhibits sprouting angiogenesis by activating DELTA-NOTCH signalingIn: Proceedings of the National Academy of Sciences of the United States of America (PNAS) , Jg. 107 2010, Nr. 28, S. 12640 – 12645
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Differential angiogenesis function of CCM2 and CCM3 in cerebral cavernous malformationsIn: Neurosurgical Focus , Jg. 29 2010, Nr. 3, S. E1
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Proliferation activity is significantly elevated in partially embolized cerebral arteriovenous malformationsIn: Cerebrovascular Diseases , Jg. 30 2010, Nr. 4, S. 396 – 401
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Susceptibility weighted magnetic resonance imaging of cerebral cavernous malformations : prospects, drawbacks, and first experience at ultra–high field strength (7-Tesla) magnetic resonance imagingIn: Neurosurgical Focus , Jg. 29 2010, Nr. 3, S. E5
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Involvement of PTEN Promoter Methylation in Cerebral Cavernous MalformationsIn: Stroke , Jg. 40 2009, Nr. 3, S. 820 – 826
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Phosphatase and tensin homolog in cerebral cavernous malformation : a potential role in pathological angiogenesisIn: Journal of Neurosurgery (JNS) , Jg. 110 2009, Nr. 3, S. 530 – 539
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Increased expression of EphA2 correlates with adverse outcome in primary and recurrent glioblastoma multiforme patientsIn: Oncology reports , Jg. 19 2008, Nr. 1, S. 151 – 156
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Implication of PTEN in production of reactive oxygen species and neuronal death in in vitro models of stroke and Parkinson's diseaseIn: Neurochemistry international , Jg. 50 2007, Nr. 3, S. 507 – 516
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Biological activity of paediatric cerebral cavernomas : an immunohistochemical study of 28 patientsIn: Child's nervous system , Jg. 22 2006, Nr. 7, S. 685 – 691
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PTEN : A crucial mediator of mitochondria-dependent apoptosisIn: Apoptosis : an international journal on programmed cell death , Jg. 11 2006, Nr. 2, S. 197 – 207
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Oleic acid causes apoptosis and dephosphorylates BadIn: Neurochemistry international , Jg. 46 2005, Nr. 2, S. 127 – 135
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Unsaturated fatty acids isolated from human lipoproteins activate protein phosphatase type 2Cβ and induce apoptosis in endothelial cellsIn: Atherosclerosis , Jg. 180 2005, Nr. 2, S. 245 – 254
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Neuroprotection by transforming growth factor-β1 involves activation of nuclear factor-κB through phosphatidylinositol-3-OH kinase/Akt and mitogen-activated protein kinase-extracellular-signal regulated kinase1,2 signaling pathwaysIn: Neuroscience , Jg. 123 2004, Nr. 4, S. 897 – 906
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Protein kinase CK2 phosphorylates BAD at threonine-117In: Neurochemistry international , Jg. 45 2004, Nr. 5, S. 747 – 752
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Cytosine arabinofuranoside-induced activation of astrocytes increases the susceptibility of neurons to glutamate due to the release of soluble factorsIn: Neurochemistry international , Jg. 42 2003, Nr. 7, S. 567 – 581
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Neuroprotection by drug-induced growth factorsIn: International congress series : ICS , Jg. 1252 2003, S. 235 – 246
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Increase in glutamate-induced neurotoxicity by activated astrocytes involves stimulation of protein kinase CIn: Journal of neurochemistry , Jg. 82 2002, Nr. 3, S. 504 – 515
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Transforming Growth Factor-β1 Increases Bad Phosphorylation and Protects Neurons Against DamageIn: The Journal of Neuroscience , Jg. 22 2002, Nr. 10, S. 3898 – 3909
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Evidence for the involvement of Par-4 in ischemic neuron cell deathIn: Journal of Cerebral Blood Flow and Metabolism (JCBFM) , Jg. 21 2001, Nr. 4, S. 334 – 343
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TGF-β1 inhibits caspase-3 activation and neuronal apoptosis in rat hippocampal culturesIn: Neurochemistry international , Jg. 38 2001, Nr. 3, S. 227 – 235
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β2-Adrenoceptor stimulation enhances latent transforming growth factor-β-binding protein-1 and transforming growth factor-β1 expression in rat hippocampus after transient forebrain ischemiaIn: Neuroscience: IBRO, International Brain Research Organization , Jg. 107 2001, Nr. 4, S. 593 – 602
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Stimulation of 5-HT1A receptors reduces apoptosis after transient forebrain ischemia in the ratIn: Brain research , Jg. 883 2000, Nr. 1, S. 41 – 50
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The expression of transforming growth factor-beta1 (TGF-β1) in hippocampal neurons : a temporary upregulated protein level after transient forebrain ischemia in the ratIn: Brain research , Jg. 866 2000, Nr. 1-2, S. 286 – 298
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The β2-adrenoceptor agonist clenbuterol modulates Bcl-2, Bcl-xl and Bax protein expression following transient forebrain ischemiaIn: Neuroscience , Jg. 90 1999, Nr. 4, S. 1255 – 1263
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β2-Adrenoceptor Agonist Clenbuterol Causes NGF Expression and NeuroprotectionIn: CNS drug reviews , Jg. 5 1999, Nr. 4, S. 347 – 364
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Lanzino G, Spetzler RF (Eds), Cavernous malformations of the brain and spinal cordIn: Neurosurgical Review , Jg. 33 2010, Nr. 2, S. 255 – 255
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Activation of NF-kB by TGF-ß1 through PI3K/Akt and MAPK/Erk1,2 signalling pathwaysIn: Pharmacology of cerebral ischemia / International Symposium on Pharmacology of Cerebral Ischemia <10, 2004, Marburg> / Krieglstein, Josef (Hrsg.) 2004, S. 397 – 405
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Mechanisms of neuroprotection by THFβ1In: Pharmacology of cerebral ischemia 2002: with 35 tables / 9th International Symposium on Pharmacology of Cerebral Ischemia, 2002, Marburg / Krieglstein, Josef (Hrsg.) 2002, S. 363 – 371
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The novel molecular pathomechanism of cerebral cavernous malformations (CCMs)Duisburg, Essen 2011